Quote:Anyone want to break this study down
Quote:The Autophagy Failure: When mTORC1 is locked in the "on" position, it paralyzes autophagy. Because neurons cannot divide to dilute trash, this autophagy failure causes a massive accumulation of toxic, misfolded proteins (like amyloid-beta plaques and tau tangles) that eventually kill the brain cells.
The same PI3K/AKT/mTORC pathway I outlined for COVID where it would help stop the virus from dividing lowering viral reproduction. Between that and an M1 macrophage shift leronlimab would work directly as an antiviral in addition to leronlimab's anti-inflammatory action. It also acts that way in cancer by helping stop tumor cells from dividing. By downregulating mTORC it could also help in Alzheimer's.