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Posted On: 01/03/2024 4:21:02 PM
Post# of 154605
ohm, I'm putting this out to you, but anyone who might know, please answer.
Dr. Lalezari has associated "blipping" with immune activation events.
So when the immune system was ramped up to take on an offender, the HIV virus would replicate.
Do you think they monitored and captured data surrounding those points? When those events occured, What caused those events to occur? What the sickness was? How high did HIV viral load climb to? Would they also have been measuring biomarkers at the time? What type of labs do you think they might have been measuring? CRP, ESR, WBC? IL-1, Interferon Gamma?
How do you think HIV was able to replicate during these events if leronlimab was still attached to ccr5?
Dr. Lalezari has associated "blipping" with immune activation events.
So when the immune system was ramped up to take on an offender, the HIV virus would replicate.
Do you think they monitored and captured data surrounding those points? When those events occured, What caused those events to occur? What the sickness was? How high did HIV viral load climb to? Would they also have been measuring biomarkers at the time? What type of labs do you think they might have been measuring? CRP, ESR, WBC? IL-1, Interferon Gamma?
How do you think HIV was able to replicate during these events if leronlimab was still attached to ccr5?
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