I have a few Questions after reading an article ye
Post# of 152090

Do we think that Leronlimab is a time saving immunotherapy drug that allows time by stopping metastasizing so our immune system can kill the tumors?
Our method of action appears to be stopping the CCR5 receptors on some cancer cells but not all cells because the CCR5 receptors are not on all cancers? Is this true?
Ohm speaks of using Leronlimab with other drugs. Is this to enhance the immune response? Or to attack the cancer cells?
Would Leronlimab work better if it was a first choice drug and not used after immune system hammering chemo?
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