Perhaps the body needs some of the CCR5 to be avai

Perhaps the body needs some of the CCR5 to be available (not 100% receptor occupancy) to perform it's normal function in order to help the liver deal with it's response to Leronlimab's effect on existing inflammation? Maybe a second or third downstream reaction to the increase in some of the positive Leronlimab effects? Seems like the only logical explanation. Although I have no idea why that would work or the MOA? I'm curious to know why Bicarbonate increased in 350mg but not 700mg. This is definitely a riddle that needs to be figured out (350mg vs 700mg).