A continuation of my last post. Although these

A continuation of my last post.

Quote:

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Although these results are important, they do not reveal the cause and effect of Alzheimer's disease. "It is still difficult to determine from human data which process triggers the disease process," says de Vries. This can only be proven by changing something in cells or animal models and observing what happens.

"That is the first thing we need to do now," stresses de Vries. "But one thing is clear. If we find the molecular basis for resilience, we will have new starting points for the development of drugs that could activate resilience-related processes in Alzheimer's patients."
https://www.focus.de/gesundheit/news/alzheime...88186.html

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The immediate cause is an inflammatory milieu. The underlying cause is either genetic factors, environmental factors or likely a combination of both. Which is a commonality in many diseases.

For the genetic factors, gene editing could help but foremost in that is a need to guard against unwanted secondary consequences.
Environmental consequences can be alleviated but it could take a century to see substantial benefit and you'd be fighting the conspiracy theorists and corporations every step of the way.

What we have for now is drugs that can reverse an over-active immune system, the accompanying inflammation and the damage it can cause. In my opinion leronlimab is the best one out there.

ps. When we get the results from the Alzheimer's pilot study someone needs to send them to de Vries.