Good article. Because these analyses were condu

Good article.

Quote:

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Because these analyses were conducted in mice and only 16 humans, the findings "call for further studies" to reveal the mechanisms that drive immune cells to the SCG, the researchers noted in the paper. This may involve studying the nerve cells that link the heart and spinal cord, as well as messenger proteins called cytokines that summon macrophages.
https://www.livescience.com/health/heart-circ...p-problems

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Chronic inflammation causes heart disease and atherosclerosis. As we age there is an increase in inflammatory factors, especially CCL5 (RANTES). So it's no wonder that the older you get the higher your risk of heart attack, Leronlimab reduces Inflammation and can reduce the plaques that can lead to atherosclerosis. In turn it would also reduce scarring of the superior cervical ganglion leading to a restored melatonin production.

By now all doctors (and those at the FDA) should realize the long list of old age related maladies caused by inflammation. They should also be well aware of CCR5s role in all of this. If a complete layman can figure this out then they should be doing everything in their effort to look at the solution (leronlimab).