Seems like the half life is only part of, maybe le

Seems like the half life is only part of, maybe less than half (pun intended) of the issue. Because when the drug is first injected or IV-ed, it begins attacking the problem, eg reducing the cytokine storm. I don't know if Leronlimab molecules are neutralized to be rendered useless, right after they attack. Half life in chemistry usually refers to the time required for half of a substance/molecule/atom to be changed or rendered useless, but usually without any outside influence, such as a virus effecting/reducing the efficacy after the attack. It's like a soldier who uses half his bullets to kill the enemy, now he has less power. The other concept would be if a soldier used his bayonet to kill the enemy, now his potential is not reduced as he kills more and more enemies.

My point is that even if the half life is shorter for IV leronlimab, why is that? Is it because it attacks the problem quicker? If yes, then the shorted half life would not really be meaningful, would not imply any inferiority, if fact, it could be a benefit.