I said early on that we just needed high doses of ACE2 Inhibitors to keep those receptors blocked so the virus can’t hitch a ride in. Everyone has hypotension but nobody dies from Covid. But since it would never be that simple I ask you, if inhibition of RAS has an effect, and if ACE2 plays a role in vital entry (this was proposed early and I honestly don’t know if that hypothesis has stuck or not) then would people already taking an ACE Inhibitor for their blood pressure have any level of protection from the virus in general and also from the effects of LH if this theory is true? I feel like I hear about ACE receptors a lot with Covid but nobody ever mentions the extremely popular meds half the country is probably on and whether or not they would have a good, bad, or neutral affect on the disease.