This is extremely intriguing. Thank you. Upon first thought, yes, pdl-1 might in fact move LL beyond oncology, HIV? Anti-PD-1s/AntiPDL-1s? Dostarlimab? Keytruda? Does ViiV/GSK or Merck know this given their deeply entrenched positions in both HIV and Cancer? Really? They had better. Otherwise, if you stumbled upon this and so did the company, then they really need to be educated on what they own. I see a patent extension from MERCK if this is true. 10 of Billion$

Yes creativ micro can measure this. Is inflammation implied in this theory?

I had to read this post 10+ times to think about the implications. WTF, My69z?

How did you stumble upon this? (Links please)

I came across LL in January 2014 searching for a cure for HIV through CCR5 blockage post-Timothy Brown (not necessarily gp120 or CXCR4 blockage) but this is ALSO including CXCR4 phenotypes??

I read it yet again…so this is turning “cold” T cells “HOT”. Has such a thing ever been described? We have cold tumors turning hot with the “priming” of LL (how, not, if is the question that plagues me) and now T-cells can turn “hot” from “cold”?

My head is spinning.

Immediate questions in my head: How or can this be applied to HIV Cure and what implications could this possibly have either HIV reservoirs?

WTF, man.

Great work! Really. My hat is off to you. I hope this is true. If you have links or sources, please post them.