Got a crazy question, but what do you think about whether it might be possible that when a patient is treated with leronlimab for some time, that leronlimab cause a tumor specific permanent change in some immune protein or some immune cell, like a memory cell or an anti-body.

What if that tumor specific particular change in immune system protein or immune system cell would be absolutely impossible to achieve unless leronlimab was initially used? What if that new tumor specific protein / anti-body is now what is circulating in the patient's body and is what is keeping the cancer at bay?

Could the blockade of CCR5 in only the way leronlimab does, induce such a permanent an specific anti-tumor change in an immune protein or cell that provides life long Immunity to that specific tumor?

And it doesn't just have to be one protein or cell, but it could be a host of proteins and cell which together, in concert, work together against the return of that tumor. But the point is that a specific anti-tumor network is somehow developed because leronlimab was used in the initial treatment of the cancerous tumor.

What I'm thinking is that something is inherently changed with the use of leronlimab and that change no longer makes it possible for that tumor to return.