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I would have to look further into the pathophysiology with each cause before I could conclude Leronlimab would help…probably a subset but I am doubtful with cardiac amyloid

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I think it would have a very good chance of clearing amyloid plaques in the heart just as it would in the brain. Both types of accumulation are based on a superstructure that involves collagen and MMP (matrix metalloproteinase) driven fibronectin. Both are also involved in fibrosis and both would be downregulated by leronlimab.