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Posted On: 10/16/2022 4:35:31 AM
Post# of 154138
Re: KenChowder #129418
Leronlimab's main mechanism is to lower inflammation and cell damage it would be independent of any variation that arises.
If my thesis that leronlimab can act as a mild anti-viral is correct it is also independent of variation since it affects the virus by inhibiting duplication.
CCR5 blockade of CCL5 disrupts the PI3K/AKT/mTOR/4E-BP1 pathway dysregulating elF4E. With elF4E disrupted the COVID-19 virus 5' -end cap would be unable to separate it's mRNA and duplicate.
If my thesis that leronlimab can act as a mild anti-viral is correct it is also independent of variation since it affects the virus by inhibiting duplication.
CCR5 blockade of CCL5 disrupts the PI3K/AKT/mTOR/4E-BP1 pathway dysregulating elF4E. With elF4E disrupted the COVID-19 virus 5' -end cap would be unable to separate it's mRNA and duplicate.
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