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Long hauler is caused by migration of cells to different parts of the body. The migrating cells have spike protein debris inside of them. When the migrating cells reach random body tissues, the immune system mounts an attack and the resulting inflammation causes long hauler symptoms.

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If long-lived monocytes encapsulating viral particles is the cause of longhaulers why aren't patients at 15-16 months going into remission?

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Patterson's long hauler theory, as expressed in this news video, is so specific and so compact that it strains credulity.

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It certainly does.

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Prescription statins reduce these effects because they prevent the migrating cells from binding to blood vessels so the migrating cells can't get into the tissues and cause inflammation.

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The binding of the monocytes to blood vessels is caused by Intercellular Adhesion Molecule 1 production of which is induced by TNF and IL-1 both of which are downregulated by a sufficient blockade of CCR5. I guess maraviroc must not be a good enough blockade if statins are being used.

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Probably Patterson's kit is the only thing that can do that on one 4 milliliter blood sample.

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Patterson's kit is a collection of already existing tests.