I ask the same question - many hospitalized CV19 patients may survive, but do they reduce greatly their viral load? Or does the body somehow build up resistance to it to cause the remission of their symptoms and the viral load stays high?

I ask this as I don't know. Thus, those with testing showing viral load reduction would be thought to be due to Brilacidin and those without reduction would be the number that would have recovered without Rems...

I personally think that with the variants now arising that the percentage of hospitalized patients dying will increase steadily with the possibility that the increase may be dramatic.

So much for my thoughts on this subject. Back to monitoring.