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Posted On: 11/14/2020 10:24:21 PM
Post# of 154898
A question for the scientists.
In HIV infection, the gp120 protein first attaches to the cd4 receptor on the cell membrane. And only then can it bind with the ccr5 receptor to facilitate cell entry.
So what happens to the HIV when the ccr5 receptor is unavailable because it is already occupied by the Leronlimab?
By that point the virus has already attached itself to the cdr4 receptor.
My understanding is that this is not a replicable process — that the virus cannot simply try again on a different cell.
And that means Leronlimab does not just inhibit viral entry; it effectively neutralizes any virus that attempts entry.
Is that correct?
In HIV infection, the gp120 protein first attaches to the cd4 receptor on the cell membrane. And only then can it bind with the ccr5 receptor to facilitate cell entry.
So what happens to the HIV when the ccr5 receptor is unavailable because it is already occupied by the Leronlimab?
By that point the virus has already attached itself to the cdr4 receptor.
My understanding is that this is not a replicable process — that the virus cannot simply try again on a different cell.
And that means Leronlimab does not just inhibit viral entry; it effectively neutralizes any virus that attempts entry.
Is that correct?
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