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Posted On: 02/24/2020 3:21:47 PM
Post# of 154119
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Washington Post
“Experts hypothesize that the difference between a lethal infection and one that feels like a bad cold probably hinges on the interaction between the virus and a person’s immune system.”
“What you get is the initial damage and rush of inflammatory cells, but the damage is so extensive that the body’s immune response is completely overwhelmed — which causes even more immune response, more immune cells and more damage,”
Bruce Patterson:
“Leronlimab enhances the innate and cellular immune response by inhibiting Treg cells which turn off the immune system. In addition, CCL5-CCR5 is one of the pathways that controls the massive migration of immune cells to sites of inflammation. Last, leronlimab reprograms macrophages. Taken together all of these effects don’t target the virus but it hopefully can mitigate the severe damage caused by the resulting cytokine storm.
Washington Post
“Experts hypothesize that the difference between a lethal infection and one that feels like a bad cold probably hinges on the interaction between the virus and a person’s immune system.”
“What you get is the initial damage and rush of inflammatory cells, but the damage is so extensive that the body’s immune response is completely overwhelmed — which causes even more immune response, more immune cells and more damage,”
Bruce Patterson:
“Leronlimab enhances the innate and cellular immune response by inhibiting Treg cells which turn off the immune system. In addition, CCL5-CCR5 is one of the pathways that controls the massive migration of immune cells to sites of inflammation. Last, leronlimab reprograms macrophages. Taken together all of these effects don’t target the virus but it hopefully can mitigate the severe damage caused by the resulting cytokine storm.
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