Yes, I agree. The guy gave few interviews since it was published until now. Very interesting. It made top news today on realclearscience. The explanation of why this works (copied below) seems logical to me, we will see what happens here.

Something like this that detects you have cancer, then say if they determined it was prostate cancer, using RP's test to determine the aggressiveness, would have great synergy. Or if they determined it was TNBC, using leronlimab much earlier than usual in the diagnosis would give even better overall survival results, because the early it is detected the better.

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No one believed it. I didn’t believe it. I thought, “Gosh, okay, maybe it’s a fluke, maybe it works just for breast cancer.” So we went on to test it in prostate cancer, which is also many different types of diseases, and it seemed to be working in all of those. We then tested it further in lymphoma. Again, many different types of lymphoma. It worked across all of those. We tested it in gastrointestinal cancer. Again, many different types, and still, it worked, but we were skeptical

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Quote:

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Well, imagine DNA as a piece of string, that’s your hard drive. Epigenetics is like the beads that you put on that string. Those beads you can take on and off as you wish and they control which apps are run, meaning which genetic programs the cell runs. We hypothesized that for cancer, those beads cluster together, rather than being randomly distributed across the string.
The implications of this are profound. It means that DNA from cancer folds in water into three-dimensional structures that are very different from healthy cells’ DNA. It’s quite literally the needle in a haystack.

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